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CD55 Polyclonal Antibody, 100ul[BT-AP01513] Antisense Oligonucleotides Heterozygous loss-of-function mutations in this

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CD55 Polyclonal Antibody, 100ul[BT-AP01513] Antisense Oligonucleotides Heterozygous loss-of-function mutations in thisCD55 encodes a glycoprotein involved in the regulation of the complement cascade. Binding of the encoded protein to complement proteins accelerates their decay, thereby disrupting the cascade and preventing damage to host cells. Antigens present on this protein constitute the Cromer blood group system (CROM). Alternative splicing results in multiple transcript variants. The predominant transcript variant encodes a membrane bound protein, but

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Description

Heterozygous loss-of-function mutations in this gene result in the dominant In(Lu) blood phenotype

Mutations in this gene are the cause of hyperlipoproteinemia| type 1D

This gene is activated during the stromal response to invasive breast cancer

It is an atypical calpain in that it lacks the calmodulin-like calcium-binding domain and instead has a divergent C-terminal domain

CD55 Polyclonal Antibody, 100ul[BT-AP01513] Antisense Oligonucleotides Heterozygous loss-of-function mutations in thisCD55 encodes a glycoprotein involved in the regulation of the complement cascade. Binding of the encoded protein to complement proteins accelerates their decay, thereby disrupting the cascade and preventing damage to host cells. Antigens present on this protein constitute the Cromer blood group system (CROM). Alternative splicing results in multiple transcript variants. The predominant transcript variant encodes a membrane bound protein, but

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